That's really great news Oracle !!!
Could you tell me what is the chemo that Lien is taking with DCA? And also, what is excactly the type of the desease is she fighting? You have probable answered these questions before, but I can't find them. If you could give me a link to an older discussion would be also fine.

My sister's cancer has metastised to the lung. There were some findings in a CT last Dec., then one spot 8mm in February, and last CT scan a week ago confirmed our fears. The 8mm spot is now 10mm, and there are about 10 more spots between 2-6 mm each. The desease progresses and I don't know what more we can do to slow it down. She has taken no chemo so far, and she is in a very good health (except of her anemia).

My impression is that DCA works, but not enough for her case, and I am searching for anything it could boost it up.

see link: http://findarticles.com/p/articles/mi_m0EIN/is_2006_June_4/ai_n16440909/

I think the biggest player was the following for Lien:

Lemon Water - lemons on everything.
the budwig diet.
alka-life -- used it on all her water including tea, coffee, soup etc.
----------------------------
mixed together - in liquid form

DCA at least 1/8 teaspoon
mixed with B1(500mg 2 tabs=1000mg)
CoQ10 400mg
magnesium 400mg (magnesium fizz)
Vit D 20,000 IU
4000mg L_GLUTAMINE
two ounces of filtered water.

- Down the Hatch! ;-0

I made Lien a pot of Black OOLONG tea broken from a pressed tea block.
I sweetened the tea with a finger full of stevia leaf and a tablespoon of fresh Honey.

This was her thing and it cleared her lymph nodes fast!!!

D - Lien and I bagged coca cola man thinking its just bad news for us. We had bone issues and loading up with carbon dioxide which is great for cancer but not for our bones. Not sure about your situation but wanted to share that knowing cancer is anaerobic,

Anaerobic Metabolism - Glycolysis
The anaerobic energy pathway, or glycolysis, creates ATP exclusively from carbohydrates, with lactic acid being a by-product. Anaerobic glycolysis provides energy by the (partial) breakdown of glucose without the need for oxygen.

Anaerobic metabolism produces energy for short, high-intensity bursts of activity lasting no more than several minutes before the lactic acid build-up reaches a threshold known as the lactate threshold and muscle pain, burning and fatigue make it difficult to maintain such intensity.

The emphasis is on "without the need for oxygen" and Lactic Acid - cancers best friend and a constant enemy to us.
The last thing we need is to make our bones weaker with carbonated beverages and to create a situation for progressive invasion.

This is of course personal experiences only being we are not doctors.

God Bless Us All

Is Lien still on low dose abraxane, or has she switched to some other chemos, and which ones?

Did she do any sort of chemofit to pick those chemos? How did you know that they are synergetic with DCA?

I have found an older post of you mentioning “chemofit”. Could you explain us more? Chemofit guided treatment is a hot issue in oncology, and I have read a lot of pros and cons about it. So far, it is not officially accepted, but many oncologists use it.

In our case, I am afraid that my sister has to do some chemo sooner or later. So, I am trying to find the best options in this area. I think that chemo, if used wisely, can give some benefits. But we need to find a good, open minded oncologist, something that is not too easy.

NOTE: Stage 4 lung cancer with metastasis to her immune system - lymph nodes, bones and liver. You can see LIENS scans on this site with the rest of them - before and after stuff.

Is Lien still on low dose abraxane, or has she switched to some other chemos, and which ones?

A - Wish she was but she is no longer on abraxane becuase it stopped working after she stopped DCA so we know it really changes how chemo acts - at least in Lien it sure does.

A -She is on Avastin, carboplatin(has been Cisplatin but stopped for her cholesterol levels) about every two three weeks.

Did she do any sort of chemofit to pick those chemos?
A - No we moved by necessity.

How did you know that they are synergistic with DCA?

A - We performed scans and the results over the past 2 and half years have proven to clearly prove that DCA definitely kicks chemo up a lot.
I believe it makes the need for high dosages of chemo smaller because it places already weak cells into a jump state and anything will pretty much kill them.

A -WHY - because Lien went into remission off of her regimented chemos for weeks at a time and when she went back on after the cancer started back up the speed at which her cancer cleared is simply unheard of.

I have found an older post of you mentioning “chemofit”. Could you explain us more? Chemofit guided treatment is a hot issue in oncology, and I have read a lot of pros and cons about it. So far, it is not officially accepted, but many oncologists use it.

In our case, I am afraid that my sister has to do some chemo sooner or later. So, I am trying to find the best options in this area. I think that chemo, if used wisely, can give some benefits. But we need to find a good, open minded oncologist, something that is not too easy.

A - comment - personal opinion only -

Stay away from any institution that offers a regular nutritionist over Naturopathic Oncology. A single nutritionist degree for heavy cancers is like hiring a radio DJ to engineer your companies sound systems. Those online degree's, or 6 months, 1, 2 or even 4 year degrees belong somewhere but I am really not sure where for heavy cancers.

I get very angry thinking back at my experience with those frightened by their own ignorance. They were not willing to admit ignorance but willing to sacrifice my wife over it.

Find a good cancer naturopathic oncologist -

- BEST ADVICE -

Contact the Cancer Treatment and Wellness Center in Renton Washington.

Setup a phone consultation with Dr. Gignac (NaturoPathic Oncologist)

Setup a phone consultation with Dr. Nick Chen (PHD Oncology)

Links:

http://www.cancercenter.com/seattle-clinic/physicians/mark-gignac.cfm
http://www.cancercenter.com/seattle-clinic/physicians.cfm

These folks are the best you will ever find at what they do. If you want to get through this I would not hesitate on getting in touch.
Not a pitch D - Lien is here because of these two people so its all real.

Good Luck D Man

God Bless Us All
O

Regarding "cholesterol levels" my sister had also total cholesterol at 270 and now is at 190 taking red yeast rice, which has also anticancer properies:
http://www.medicalnewstoday.com/articles/110482.php

I see a new post here "Chemo, DCA, and Alternative response" but there is some error in the server and we can't read it. I hope the administrator could take care of it.

I would add (for those of you who don't know) that statins affect a pathway in the liver that generates choloresterol and shutting down this pathway also shuts down production of co-enzyme q10 so if you are going to use statins or red rice you should supplement with co-enzyme q10. This may be especially significant in breast cancer where there are studies that indicate patients have much lower levels of CoQ10.

Statins also have anti-inflammatory properties which some think may be their real health benefit- especially in cancer where inflammation drives growth and spread. However their use in cancer is controversial- some studies claimed it reduced chance of cancer, others say it increased it- some went further and said specifically long term use of statins in obese men increased the risk of prostate cancer so benefits may differ between cancer types and other confounding factors. Perhaps the risk to obese men in long term use related to creating low levels of coenzyme q10??? In any case Co-Q10 supplementation with statin thereapy for whtever reason shouldn't hurt and may help.

Mack

Regarding statin drugs, in a study for prostate cancer on mice, RYR showed much better performance than lovostatin. This shows that there is more into RYR.

http://www.cancermonthly.com/cancerwire/january2009.html
"Dr. Hong and her colleagues compared the drug lovastatin to red yeast rice on both androgen- dependent and androgen-independent prostate cancer cells. The lovastatin decreased prostate cancer cell growth by 20 percent in the androgen- dependent cancer cells, and by 15 percent in the androgen-independent cells. The same dose of red yeast rice inhibited androgen-dependent prostate cancer cells by 47 percent, and androgen- independent cells by 62 percent. When treatment was extended for another day, red yeast rice inhibited prostate cancer cell growth even more - by 77 percent and 65 percent, respectively.
Even a form of red yeast rice that did not contain monacolin K reduced prostate cancer growth, suggesting that red yeast rice contains other components that are active against cancer cells. Among these components are red pigments, which are thought to have anti-cancer effects"

Thought I'd do a quick latest pubmed search on statins and cancer and see what came up. These are not Red Rice- all the artifical ones. I want to look into Red Rice Separately to find more info now. I assume it is one of those treatments the FDA has banned from use?

What seems apparent is statin effectiveness depends on the type of cancer and type of statin. Of interest is the good effect of the fat loving statins against gynecological cancers that express the very thing statins inhibit but I don't know if there is an available test for which other cancers may express HMGCoA.

Mack

Statins inhibit Akt/PKB signaling via P2X7 receptor in pancreatic cancer cells.

Lipophilic but not hydrophilic statins selectively induce cell death in gynecological cancers expressing high levels of HMGCoA reductase.

Currently, scientific data are slightly in favor of the notion that statins do not cause a significant increase in cancer rates. However, there are many studies that show small correlations between statin use and increased incidence of cancer and therefore, we feel further prospective studies are needed. Whether statins do cause cancer remains uncertain at this point in time.

Immunomodulatory properties of statins and cancer risk

Statins inhibit the growth of variant human embryonic stem cells and cancer cells in vitro but not normal human embryonic stem cells.

Simvastatin and lovastatin inhibit breast cell invasion induced by H-Ras.

Combined Treatment of {gamma}-Tocotrienol with Statins Induce Mammary Tumor Cell Cycle Arrest in G1.

Red rice/Sesame seeds raw/Salt

Pan fry the sesame seeds (1/3 cup) on low heat until shiny and aromatic (about 5 to 6 minutes). Leave in the pan to cool with a teatowel on top. Apparently come back to it 2 hours later and grind it up with a mortar/pestle to a medium coarse consistency. Not too fine. To every 12 teaspoons of sesame seeds add one teaspoon of salt.

Then steam some red rice in a steamer The little bowl where the rice is in has to have a hole in the lid and then the whole thing is covered by a lid.(?? I don't steam much food so is this some special kind of steamer ??) Put one cup of rice into the bowl inside the steamer with two to 2.5 cups of water. Steam for 2 hours until the rice is soft. Sprinkle with a little bit of the sesame seed mixture. (You can keep the sesame seed mixture stored up for later sprinkles onto more rice). Chew it in the mouth until it is a milky paste and then swallow.

Some obvious questions: why the sesame seeds? Why the salt? Do liver cancers overexpress HMGCoA and hence response to Statins??? Always more questions...

Mack

Links to reports in previous email now included below:

Statins inhibit Akt/PKB signaling via P2X7 receptor in
pancreatic cancer cells.
http://www.ncbi.nlm.nih.gov/pubmed/19540829?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Lipophilic but not hydrophilic statins selectively induce
cell death in gynecological cancers expressing high levels
of HMGCoA
reductase.
http://www.ncbi.nlm.nih.gov/pubmed/19432822?ordinalpos=16&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Currently, scientific data are slightly in favor of the
notion that statins do not cause a significant increase in
cancer rates. However, there are many studies that show
small correlations between statin use and increased
incidence of cancer and therefore, we feel further
prospective studies are needed. Whether statins do cause
cancer remains uncertain at this point in time.
[http://www.ncbi.nlm.nih.gov/pubmed/19451806?ordinalpos=10&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Immunomodulatory properties of statins and cancer risk
http://www.ncbi.nlm.nih.gov/pubmed/19445281?ordinalpos=12&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Statins inhibit the growth of variant human embryonic stem
cells and cancer cells in vitro but not normal human embryonic stem cells.
http://www.ncbi.nlm.nih.gov/pubmed/19438511?ordinalpos=14&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Simvastatin and lovastatin inhibit breast cell invasion
induced by H-Ras.
http://www.ncbi.nlm.nih.gov/pubmed/19360310?ordinalpos=26&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

Combined Treatment of {gamma}-Tocotrienol with Statins
Induce Mammary Tumor Cell Cycle Arrest in G1.
http://www.ncbi.nlm.nih.gov/pubmed/19359655?ordinalpos=27&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DefaultReportPanel.Pubmed_RVDocSum

And also:
Synergistic antiproliferative effects of gamma-tocotrienol and statin treatment on mammary tumor cells.
http://www.ncbi.nlm.nih.gov/pubmed/17701065?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DiscoveryPanel.Pubmed_Discovery_RA&linkpos=1&log$=relatedarticles&logdbfrom=pubmed

So, I ordered gamma-tocotrienol from iherb: http://www.iherb.com/Twinlab-MaxiLife-Rice-Tocotrienols-50-mg-60-Softgels/4384?at=0
I hope there is a synergistic effect with RYR